Biochemistry
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Modelling the active SARS-CoV-2 helicase complex as a basis for structure-based inhibitor design
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Conformational diversity of CDR region during affinity maturation determines the affinity and stability of Sars-Cov-1 VHH-72 nanobody
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Remdesivir is a delayed translocation inhibitor of SARS CoV-2 replication in vitro
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Designed Variants of ACE2-Fc that Decouple Anti-SARS-CoV-2 Activities from Unwanted Cardiovascular Effects
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High Affinity Nanobodies Block SARS-CoV-2 Spike Receptor Binding Domain Interaction with Human Angiotensin Converting Enzyme
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SARS-CoV-2 spike-glycoprotein processing at S1/S2 and S2’and shedding of the ACE2 viral receptor: roles of Furin and TMPRSS2 and implications for viral infectivity and cell-to-cell fusion
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Potent in vitro anti-SARS-CoV-2 activity by gallinamide A and analogues via inhibition of cathepsin L
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Structural basis for the inhibition of the papain-like protease of SARS-CoV-2 by small molecules
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Mechanism of SARS-CoV-2 polymerase inhibition by remdesivir
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TMPRSS2 and ADAM17 interactions with ACE2 complexed with SARS-CoV-2 and B 0 AT1 putatively in intestine, cardiomyocytes, and kidney
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